Typical Event Horizons in AdS/CFT

We consider the construction of local bulk operators in a black hole background dual to a pure state in conformal field theory. The properties of these operators in a microcanonical ensemble are studied. It has been argued in the literature that typical states in such an ensemble contain firewalls, or otherwise singular horizons. We argue this conclusion can be avoided with a proper definition of the interior operators.Comment: 8 pages, 2 figures. arXiv admin note: text overlap with arXiv:1310.799

Techniques for studying gravity waves and turbulence: Horizontal, vertical and temporal resolution needed

One of the most important atmospheric measurements that is needed is a measure of the gravity-wave spectrum. The MST radar has been investigated as means to measure the temporal resolution required to determine gravity-wave oscillations. The required vertical and horizontal resolution is dependent on the particular part of the gravity wave spectrum that is analyzed. Horizontal spacing is also discussed

Forage Feeding Systems for Dairy Cattle

PDF pages: 1

Experimental aerodynamic heating to simulated shuttle tiles

The heat transfer to simulated shuttle thermal protection system tiles was investigated experimentally using a highly instrumented metallic thin wall tile arranged with other metal tiles in a staggered tile array. Cold-wall heating rate data for laminar and turbulent flow were obtained in the Langley 8-foot high temperature tunnel at a nominal Mach number of 7, a nominal total temperature of 3300 R, free-stream unit Reynolds number from 3.4 x 10 to the 5th power to 2.2 x 10 to the 6th power per foot, and free-stream dynamic pressure of 1.8 psia to 9.1 psia. Experimental data are presented to illustrate the effects of flow angularity and gap width on both local peak heating and overall heating loads

A program continuation to develop processing procedures for advanced silicon solar cells

Shallow junctions, aluminum back surface fields and tantalum pentoxide (Ta205) antireflection coatings coupled with the development of a chromium-palladium-silver contact system, were used to produce a 2 x 4 cm wraparound contact silicon solar cell. One thousand cells were successfully fabricated using batch processing techniques. These cells were 0.020 mm thick, with the majority (800) made from nominal ten ohm-cm silicon and the remainder from nominal 30 ohm-cm material. Unfiltered, these cells delivered a minimum AMO efficiency at 25 C of 11.5 percent and successfully passed all the normal in-process and acceptance tests required for space flight cells

Identifying safety strategies for on-farm grain bins using risk analysis

The potential for grain bin accidents exists each year on Arkansas farms and farms across the nation. The trend toward increasing utilization of on-farm grain drying and storage could lead to an increase in grain bin accidents. The sharp contrast between a safe, efficient operation and one that leads to injury or death can be represented as sets of farmer-decisions and subsequent chance events. A model was constructed to define the risk associated with grain bin entry and inbin activity so that safety interventions could be identified and implemented to reduce the probability of injury and death. A survey was distributed to Arkansas grain farmers to gather data on the level of safety education, storage techniques, operations management, and other parameters. The data collected from the survey provided quantitative input of many of the model’s probability-distribution functions. Using a fault tree (with parallel modes of failure) in conjunction with a Monte Carlo simulation technique, we evaluated six safety intervention strategies and identified the one with the greatest potential for reducing the risk of serous injury or death. As part of senior design in biological engineering, plans are underway to design and test a probe that can locate and break bridged grain (a common risk factor in grain bin management) while working outside the bin on the ground

The Cosmic-ray Neutron Probe Method for Estimating Field Scale Soil Water Content: Advances and Applications

The need for accurate, real-time, reliable, and multi-scale soil water content (SWC) monitoring is critical for a multitude of scientific disciplines trying to understand and predict the earth’s terrestrial energy, water, and nutrient cycles. One promising technique to help meet this demand is fixed and roving cosmic-ray neutron probes (CRNP). However, the relationship between observed low-energy neutrons and SWC is affected by local soil and vegetation calibration parameters. This effect may be accounted for by a calibrated equation based on local soil type and the amount of standing biomass. However, determining the calibration parameters for this equation is labor and time intensive, thus limiting the full potential of the roving CRNP in large surveys and long transects, or its use in novel environments. In this work, our objective is to develop and test the accuracy of using globally available datasets (clay weight percent, soil bulk density, and soil organic carbon) to support the operability of the CRNP. Here, we develop a 1 km product of soil lattice water over the CONtinental United States (CONUS) using a database of in-situ calibration samples and globally available soil taxonomy and soil texture data. We then test the accuracy of the global dataset in the CONUS using comparisons of 61 in-situ samples of clay percent (RMSE = 5.45 wt. %, R2 = 0.68), soil bulk density (RMSE = 0.173 g/cm3, R2 = 0.203), and soil organic carbon (RMSE = 1.47 wt. %, R2 = 0.175). In addition, we conduct an uncertainty analysis of the global soil calibration parameters using a Monte Carlo error propagation analysis (maximum RSME ~0.035 cm3/cm3 at a SWC = 0.40 cm3/cm3). Fast growing crops (i.e. maize and soybeans) contribute to the CRNP signal primarily through the water within their biomass and this signal must be minimized for soil moisture retrieval. This was done by using a vegetation index derived from MODIS imagery as a proxy for standing wet biomass (RMSE \u3c 1 kg/m2). Lastly, we make recommendations to the design and validation of future roving CRNP experiments. Advisor: Trenton E. Fran

Axon Death Prevented: Wld\u3csup\u3es\u3c/sup\u3e and Other Neuroprotective Molecules: A Dissertation

A common feature of many neuropathies is axon degeneration. While the reasons for degeneration differ greatly, the process of degeneration itself is similar in most cases. Axon degeneration after axotomy is termed ‘Wallerian degeneration,’ whereby injured axons rapidly fragment and disappear after a short period of latency (Waller, 1850). Wallerian degeneration was thought to be a passive process until the discovery of the Wallerian degeneration slow (Wlds) mouse mutant. In these mice, axons survive and function for weeks after nerve transection. Furthermore, when the full-length protein is inserted into mouse models of disease with an axon degeneration phenotype (such as progressive motor neuronopathy), Wlds is able to delay disease onset (for a review, see Coleman, 2005). Wlds has been cloned and was found to be a fusion event of two neighboring genes: Ube4b, which encodes an ubiquitinating enzyme, and NMNAT-1 (nicotinamide mononucleotide adenylyltransferase-1), which encodes a key factor in NAD (nicotinamide adenine dinucleotide) biosynthesis, joined by a 54 nucleotide linker span (Mack et al., 2001). To address the role of Wlds domains in axon protection and to characterize the subcellular localization of Wlds in neurons, our lab developed a novel method to study Wallerian degeneration in Drosophila in vivo (MacDonald et al., 2006). Using this method, we have discovered that mouse Wlds can also protect Drosophila axons for weeks after acute injury, indicating that the molecular mechanisms of Wallerian degeneration are well conserved between mouse and Drosophila. This observation allows us to use an easily manipulated genetic model to move the Wlds field forward; we can readily identify what Wlds domains give the greatest protection after injury and where in the neuron protection occurs. In chapter two of this thesis, I identify the minimal domains of Wlds that are needed for protection of severed Drosophila axons: the first 16 amino acids of Ube4b fused to Nmnat1. Although Nmnat1 and Wlds are nuclear proteins, we find evidence of a non-nuclear role in axonal protection in that a mitochondrial protein, Nmnat3, protects axons as well as Wlds. In chapter 3, I further explore a role for mitochondria in Wlds-mediated severed axon protection and find the first cell biological changes seen in a Wlds-expressing neuron. The mitochondria of Wlds- and Nmnat3-expressing neurons are more motile before injury. We find this motility is necessary for protection as suppressing the motility with miro heterozygous alleles suppresses Wldsmediated axon protection. We also find that Wlds- and Nmnat3- expressing neurons show a decrease in calcium fluorescent reporter, gCaMP3, signal after axotomy. We propose a model whereby Wlds, through production of NAD in the mitochondria, leads to an increase in calcium buffering capacity, which would decrease the amount of calcium in the cytosol, allowing for more motile mitochondria. In the case of injury, the high calcium signal is buffered more quickly and so cannot signal for the axon to die. Finally, in chapter 4 of my thesis, I identify a gene in an EMS-based forward genetic screen which can suppress Wallerian degeneration. This mutant is a loss of function, which, for the first time, definitively demonstrates that Wallerian degeneration is an active process. The mammalian homologue of the gene encodes a mitochondrial protein, which in light of the rest of the work in this thesis, highlights the importance of mitochondria in neuronal health and disease. In conclusion, the work presented in this thesis highlights a role for mitochondria in both Wlds-mediated axon protection and Wallerian degeneration itself. I identified the first cell biological changes seen in Wlds-expressing neurons and show that at least one of these is necessary for its protection of severed axons. I also helped find the first Wallerian degeneration loss-of-function mutant, showing Wallerian degeneration is an active process, mediated by a molecularly distinct axonal degeneration pathway. The future of the axon degeneration field should focus on the mitochondria as a potential therapeutic target